Skip Navigation

This Article
Right arrow Full Text (PDF)
Right arrow E-Letters: Submit a response to the article
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by INADA, T.
Right arrow Articles by KURODA, M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by INADA, T.
Right arrow Articles by KURODA, M.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

British Journal of Anaesthesia, 1995, Vol. 74, No. 3 283-286
© 1995 The Board of Management and Trustees of the British Journal of Anaesthesia

research-article

Tracheal tube cuff pressure during cardiac surgery using cardiopulmonary bypass

T. INADA, MD, S. KAWACHI, MD and M. KURODA, MD

Department of Anaesthesia, Matsue Red Cross Hospital 200 Horomachi, Matsue-city, Shimane 690, Japan.

Correspondence to T.I.

To determine the effects of cardiopulmonary bypass (CPB) on tracheal cuff pressure, we have measured intracuff pressure (ICP) in 29 consecutive patients undergoing cardiac surgery with CPB. Premedication comprised hyoscine and, after induction of anaesthesia with diazepam and fentanyl, followed by vecuronium, the trachea was intubated using a Portex Profile tracheal tube. Anaesthesia was maintained with high-dose fentanyl and 100% oxygen. ICP was measured with a transducer and the ICP was adjusted to 20 mm Hg. CPB was used with mild to deep hypothermia and blood-gas tensions were regulated according to alpha-stat (temperature uncorrected) pH management. Before CPB, ICP was significantly reduced from the mean baseline value of 20 (SEM 0.2) to 16.7 (0.6) mm Hg (P < 0.01). ICP changed significantly during CPB, decreasing to 8.0 (1.0) mm Hg before rewarming {P < 0.01 vs immediately before CPB) and increasing to 17.0 (0.6) mm Hg after the start of rewarming (P < 0.01 vs before rewarming). After CPB, ICP did not differ significantly from that immediately before CPB. We conclude that the decrease in ICP during the hypothermic phase of CPB may protect the tracheal mucosa against hypotensive ischaemic injury. (Br. J. Anaesth. 1995; 74: 283–286)


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?




Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.