Skip Navigation

This Article
Right arrow Full Text (PDF)
Right arrow E-Letters: Submit a response to the article
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (72)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by LEWIS, G. B. H.
Right arrow Articles by HECKER, J. F.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by LEWIS, G. B. H.
Right arrow Articles by HECKER, J. F.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

British Journal of Anaesthesia, 1985, Vol. 57, No. 2 220-233
© 1985 The Board of Management and Trustees of the British Journal of Anaesthesia

review-article

INFUSION THROMBOPHLEBITIS

G. B. H. LEWIS, M.B., B.S., M.LITT., D.A., F.F.Å.R.A.C.S. and J. F. HECKER, B.SC., B.V.SC., PH.D.

Department of Physiology, The University of New England Armidale, 2350 New South Wales, Australia

Infusion thrombophlebitis is a common complication of i.v. infusions. Many factors appear to be involved in its aetiology, of which the duration of infusion, the drugs infused and the solution(s) infused are the most important. Effective prophylaxis should be based on an understanding of the possible pathophysiology.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 J Antimicrob ChemotherHome page
P. Lanbeck, I. Odenholt, and K. Riesbeck
Dicloxacillin and erythromycin at high concentrations increase ICAM-1 expression by endothelial cells: a possible factor in the pathogenesis of infusion phlebitis
J. Antimicrob. Chemother., February 1, 2004; 53(2): 174 - 179.
[Abstract] [Full Text] [PDF]

Home page
 Postgrad. Med. J.Home page
C Waitt, P Waitt, and M Pirmohamed
Intravenous therapy
Postgrad. Med. J., January 1, 2004; 80(939): 1 - 6.
[Abstract] [Full Text] [PDF]

Home page
 ANGIOLOGYHome page
N. Hatori, M. Shimizu, H. Yoshizu, D. Segawa, T. Kimura, Y. Iizuka, S. Tanaka, and M. Fujita
Neutralization of Prostaglandin E1 Intravenous Solution Reduces Infusion Phlebitis
Angiology, September 1, 2000; 51(9): 719 - 723.
[Abstract] [PDF]

Home page
 ChestHome page
A. Pose-Reino, J. M. Taboada-Coton, D. Alvarez, J. Suarez, and L. Valdes
Infusion Phlebitis in Patients in a General Internal Medicine Service
Chest, June 1, 2000; 117(6): 1822 - 1823.
[Full Text] [PDF]

Home page
 ChestHome page
M. Monreal, F. Quilez, C. Rey-Joly, S. Rodriguez, N. Sopena, C. Neira, and J. Roca
Infusion Phlebitis in Patients With Acute Pneumonia: A Prospective Study
Chest, June 1, 1999; 115(6): 1576 - 1580.
[Abstract] [Full Text] [PDF]

Home page
 Arch Pediatr Adolesc MedHome page
D. B. Nelson and J. S. Garland
The Natural History of Teflon Catheter-Associated Phlebitis in Children
Arch Pediatr Adolesc Med, October 1, 1987; 141(10): 1090 - 1094.
[Abstract] [PDF]


Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.